‘Sometimes when you are standing outside on a dark night, you may see a “shooting star.” But, is it really a star? NO! It is a random space rock speeding through our atmosphere, burning up with tremendous frictional heat, causing light to streak across the sky. If it is a large enough chunk of rock, it will not entirely burn up, but hit the earth and make an impact crater. How many impact craters are here on Earth? It is estimated that there are about 100. How many impact craters are on the moon? There are 5,185 moon craters that are more than 12 miles across, but if you count the craters less than one-half mile in diameter, it brings the total to about one million! Why are there more craters on the moon than on Earth?
Earth has an atmosphere that burns up space rocks; the moon does not. Without our atmosphere, rocks could be routinely plummeting through our homes at night. Fortunately, our atmosphere is almost 50 miles thick, and as space rocks encounter it, friction is produced, and rocks are disintegrated. Did our atmosphere develop by chance over millions of years? If so, we should find billions of meteorites buried within the rock layers of the earth. We do not. Our atmosphere was set up from the beginning to protect life on Earth. It did not happen by accident and chance; our atmosphere was created by God to protect us! So as you “wish upon a falling star,” thank the One who made it all possible.’ From an email sent by http://www.searchforthetruth.net/
“Instagram did take our feed post down. This is ok, fortunately we have talked with our team at YouTube and they’re keeping the documentary up there, which is most important. They did demonetise and take the video out of the algorithm. Which is all ok, we assumed this would happen,” said Cole in an update about the video.”
“The biggest thing this does is significantly reduce the video’s reach. The more people the video reaches, the more people who can find help. This makes it where you can really only watch the video if you have a link or go directly to our channel.”
“YouTube won’t further share it,” Cole continued. “At this point we’ll leave the message in God’s hands and trust that whoever is supposed to watch it, will watch it. You guys have supported this so much and we’re so thankful. If you feel lead, please share the documentary with people you know.”
BACKGROUND:
Husband Cole and wife Savannah together have over 10 million Instagram followers and 13 million YouTube, subscribers.
‘Tennessee GOP Gov. Bill Lee has invited the private, conservative Hillsdale College to help open charter schools in his state as alternatives to public school that families and others in the state believe have far too liberal curricula.
Lee is making available as much as $32 million in public funds for the charter schools, which receive taxpayer funding to operate but are privately run.
They have traditionally served as an alternative to families with children in under-performing public schools.
The Hillsdale charter schools are neither owned nor managed by Hillsdale. Instead, the schools enter agreements to use the Hillsdale curriculum, and the small Michigan college provides training for faculty and staff, as well as other assistance – all free of charge, according to The New York Times.
A Hillsdale official told Just the News on Thursday it has received three applications to participate in the Tennessee program.
Hillsdale calls its lessons the “1776 Curriculum.”
School officials say the name is not in response to The New York Times’ “1619 Project” about so-called Critical Race Theory – which suggests America is inherently racist – but is “inspired by a deep admiration and respect for America’s Founders and the principles they expressed.
Public school parents have in recent months expressed large concerns about variations of critical race theory being taught to their children and have repeatedly brought their arguments to the public forum – including open school board meetings.
Hillsdale’s version teaches students that America is “an exceptionally good country.”
Critics of the 1776 Curriculum say it has an overly positive take on American history.
“It talks about the enormity of slavery, but in almost every case, everything that’s bad about America will be undone by what is good. Almost, literally, that American ideals will overcome whatever evils may be there,” Sean Wilentz, a Princeton professor told The Times.
‘We know that SARS-CoV-2 is a man-made “paravirus” if you will, created in Wuhan/Moderna laboratories and reinforced by mainstream media propaganda. But in the grand scheme, at least the first iteration released onto the world, so-called COVID-19 is a lightweight illness that is mostly just rebranded influenza.
The mRNA and viral vector injections, along with Remdesivir, combine for a quick two-year, $200 billion global racket for big pharma and Bill Gates. Ivermectin is a proven, powerful drug to treat and prevent so-called COVID-19, according to 108 peer-reviewed studies. The Ivermectin Merck patents are long expired. So the cheap, $1-per-dose, Nobel Prize-winning drug poses a serious threat not only to the emergency use authorizations for the lethal injections, but also to the temporary COVID-19 racket. But those simply cannot be the only reasons for the persistent, petulant, childish mainstream media anti-Ivermectin propaganda.
This blogger has seen scattered studies concluding that Ivermectin not only inhibits cancer cell growth, but also kills cancer cells. Perhaps placing nine said studies into one article can help disrupt the cancer industrial complex and wake up the snoozing masses.
1) American Journal of Cancer Research – 2018
This study by researchers at Unidad de Investigación Biomédica en Cáncer in Mexico concluded:
So far, at least 235 clinically-approved, non-cancer drugs have proven anti-tumor activity either in vitro, in vivo, or even clinically. Among these, ivermectin, an anti-parasitic compound of wide use in veterinary and human medicine, is clearly a strong candidate for repositioning, based on the fact that:
i) it is very safe, causing almost no side-effects other than those caused by the immune and inflammatory responses against the parasite in infected patients, and
ii) it has proven anti-tumor activity in pre-clinical studies. On the other hand, it is now evident that the use of very selective “unitargeted” drugs is commonly associated to early development of resistance by cancer cells, hence the use of “dirty” or “multitargeted” drugs is important to explore.
Some key findings by Chinese researchers at Bengbu Medical College include the following:
Recent studies have also found that Ivermectin (IVM) could promote the death of tumor cells by regulating the tumor micro-environment in breast cancer.
In an experiment designed to screen potential drugs for the treatment of leukemia, IVM preferentially killed leukemia cells at low concentrations without affecting normal hematopoietic cells.
In a study by Hashimoto, it found that IVM inhibited the proliferation of various ovarian cancer cell lines.
Experiments confirmed that IVM could significantly inhibit the proliferation of five renal cell carcinoma cell lines without affecting the proliferation of normal kidney cells, and its mechanism may be related to the induction of mitochondrial dysfunction.
Researchers at Henan University in China concluded:
We have demonstrated that ivermectin may regulate the expression of crucial molecules Caspase-3, Bax, Bcl-2, PARP, and Cleaved-PARP in the apoptosis pathway by increasing ROS production and inhibiting the cell cycle in the S phase to inhibit colorectal cancer cells (Figure 11). Therefore, current results indicate that ivermectin might be a new potential anticancer drug for treating human colorectal cancer and other cancers.
Researchers at the National Cancer Institute in Mexico City concluded the following:
Results from the present study demonstrated that ivermectin preferentially targeted the stem cell population in MDA–MB–231 human breast cancer cells. Ivermectin has been demonstrated to be safe, following treatment of millions of patients with onchocerciasis and other parasitic diseases, which makes it a strong candidate for further studies investigating its potential use as a repurposed drug for cancer therapy.
Researchers at three Chinese institutions concluded:
Those findings provided the potential targeted lncRNA-EIF4A3-mRNA pathways of ivermectin in ovarian cancer, and constructed the effective prognostic model, which benefits discovery of novel mechanism of ivermectin to suppress ovarian cancer cells, and the ivermectin-related molecule-panel changes benefit for its personalized drug therapy and prognostic assessment towards its predictive, preventive, and personalized medicine (PPPM) in ovarian cancers.
Chinese researchers at Henan University, concluded the following:
We demonstrated that ivermectin effectively inhibit the proliferation of esophageal squamous cell carcinoma (ESCC) cells by inducing mitochondrial dysfunction, suppressing NF-κB signaling and promoting apoptosis. Our results suggest that ivermectin may be a potential therapeutic target against ESCC.
Some key findings from researchers at Instituto Nacional de Cancerologia in Mexico City:
Ivermectin reduced both cell viability and colony formation capacity in the stem cell-enriched population as compared with the parental one. Finally, in tumor-bearing mice ivermectin successfully reduced both tumor size and weight. Our results on the anti-tumor effects of ivermectin support its clinical testing.
Some key findings by University of Geneva researchers are as follows:
Constitutive activation of canonical WNT-TCF signaling is implicated in multiple diseases, including intestine and lung cancers, but there are no WNT-TCF antagonists in clinical use. We report that Ivermectin inhibits the expression of WNT-TCF targets, mimicking dnTCF, and that its low concentration effects are rescued by direct activation by TCFVP16.
In vivo, Ivermectin selectively inhibits TCF-dependent, but not TCF-independent, xenograft growth without obvious side effects. Given that Ivermectin is a safe anti-parasitic agent used by 200 million people against river blindness, our results suggest its additional use as a therapeutic WNT-TCF pathway response blocker to treat WNT-TCF-dependent diseases including multiple cancers.
Job 38:4 “Where wast thou when I laid the foundations of the earth? declare, if thou hast understanding.”
‘Every day, all over the Earth, plants engage in chemical warfare against insects and animals that would eat them. When this drama is described by evolutionary scientists, they usually talk about plants as though the plants were skilled chemists who developed their abilities on their own.
For example, the leaves of the oak tree contain tannins. Tannins form complexes with proteins so that, when eaten, they have little nutritional value. An evolutionary account of how this arrangement came about describes how the trees supposedly developed this strategy for self-defense. Stories like this make one wonder how oak trees gained so much knowledge about animal digestion and chemistry. Some species of milkweed and dogbane produce powerful muscle relaxants that can be fatal to humans.
One might picture, in the far distant past, white-smocked milkweeds working in the chemistry lab. Other “doctor” milkweeds are feeding various concoctions to humans in cages to test their responses. When one tries to account for this without a Creator, the picture can become silly.
The Bible provides an answer that makes a great deal more sense. In Job 38 and 39 the Lord – the Creator – asks Job about dozens of aspects of the creation. In His questioning, He asks Job about the source of the knowledge and abilities found in living things. The answer, of course, is that the creating God, and no one else, designed, built and taught the creation. Other answers simply can’t satisfy.’https://creationmoments.com/sermons/plant-self-defense-3/?mc_cid=90ceddb138&mc_eid=00c1dcff3c
